Sgt. Terrance Yeakey

yeak

h/t Vulture of Critique

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Feds Pile New Charges on Top Alien Smuggler

Feds Pile New Charges on Top Alien Smuggler

by DC Dave

Press Quiet about It

Stan ncga

Stan Eury

On September 30, 2014, led by representatives of President Obama’s Justice Department in Greensboro, North Carolina, a federal grand jury issued an 87-count indictment of Stan Eury, the man that Mother Jones magazine once described as the leader of “the largest alien smuggling ring in our nation’s history.” It supersedes the 41-count indictment issued against Eury, his daughter, Sara Elizabeth Farrell, and the company that Farrell ostensibly directed, International Labor Management Company (ILMC), on January 31 of this year.

Most notable, in addition to the 46 additional counts on the indictment is that Eury’s daughter Farrell, who was the object of 40 of the 41 original counts, has now been dropped from the indictment entirely and has been replaced by Eury’s top lieutenants, Lee Wicker and Ken White. This bears out what we had heard weeks ago from sources within the North Carolina Employment Security Commission (NCESC), the state agency that oversees the farm and temporary employment programs that the three men are charged with abusing, that Farrell had reached a plea agreement with the prosecutors. Her name appears nowhere in the new indictment but her initials, S.E.F., appear repeatedly as a cooperating witness. In street parlance, she has “flipped” and is now singing like a canary.

lee wicker ncga(1)(1)

Lee Wicker

The “legal” alien importing empire that Eury and White began building in 1989 after they were fired from their NCESC jobs for marijuana growing would appear to be on the verge of complete collapse (Wicker is also an NCESC alumnus.). In my April 17 article “AP Gives Alien Smugglers ‘Infomercial’” I surmised that Wicker was heir apparent to an alien smuggling ring exploiting the H-2A and H-2B guest-worker programs that would continue to rock along as if nothing had happened, with Eury knocked off the perch at the top. I had arrived at that conclusion by reading the tealeaves of the media coverage. None of the major newspapers in North Carolina had even reported the January indictment, as I noted in my article, “Has Obama Gone Bulworth on Alien Smuggling?” Then most of them had run the AP story still singing the praises of Eury’s North Carolina Growers’ Association (NCGA), but with no mention of Eury or his indictment.

As the new indictment, shows, however, the stench emanating from the Eury’s operation was even greater than my sources in NCESC even knew about, and it was apparently too much for the federal prosecutors. My sources did not know that Eury and his associates had set up shell companies going by the initials CSI, ASAP, and TLC that they claimed were making financial demands upon them. Through such self-dealing, in addition to abusing the guest worker program in the various ways detailed in the earlier indictment, they also managed to extort money from farmers in the NCGA, claiming that if the fees were not paid to these phony companies the farmers would not get, or would lose their workers. It is all laid out in the section of the indictment entitled “Scheme and Artifice to Defraud.” Paragraph #25 under that section is the following:

It was a further part of the scheme and artifice to defraud that, from on or about February 12, 2009, to on or about December 31, 2013, the exact dates to the Grand Jurors unknown, that ASAP received approximately $5,561, 669.00 in payments from ILMC and TLC clients under the pretense that ASAP was collecting a $99.00 per-worker recruiting and processing fee for CSI in Mexico.

Big-money1

It is alleged in the indictment that a good bit of that money and other ill-gotten lucre ended up in the personal bank accounts of the three men, but the numbers tell a further story. The sum of $5,561,669 raised by way of a fee of $99 per recruited worker means that 56,178 workers were brought in over the slightly less than five-year period. That works out to something more than 11,235 workers brought in yearly, and we have no idea how many of them went back to Mexico. There is no mechanism set up to make sure that they go back, and there is no way of counting those who stay and those who return. If you’re wondering where all those illegal aliens in the country came from, as the Mother Jones article alluded to earlier suggests, here’s a good place to start.

It’s even worse than the aggregate numbers show, according to my NCESC sources. In spite of increasing mechanization causing a decline in demand for agricultural labor, the trend in approved work orders for the NCGA has been steadily up. Most surprising of all, in spite of the late January indictment of Eury and his daughter, NCESC approval of NCGA work orders this year have reached a record high at around 15,000 workers. The tension created by the importation of workers for whom there are no jobs is shown in my July 29 article, “Video of Labor Goon Slugging Labor Organizer.” The “labor goon” in the title was one of Eury’s.

Media-Blackout

Media Blackout

Maybe the folks at the NCESC who keep on approving NCESC orders for more and more guest workers in spite of the indictments were reading the same media tealeaves that I was. The criminal assault on the labor organizer managed to make it onto the popular left-liberal web site, Buzzfeed, but it went completely unreported in North Carolina’s major media. Now almost two weeks have passed since this latest blockbuster came down on the backs of Stan Eury and his buddies and not a peep has been uttered by anyone in the news media. The copy of the indictment that I have posted here was leaked to a contact of mine in the NCESC. The contact has been sworn to secrecy about the source of the leak, and I am unable to divulge who my contact is.

As the Obama Justice Department forges ahead in its pursuit of a man who has been coddled and even celebrated by the mainstream media—which we can’t help but believe is because he has furthered the agenda of the powers that be to break down the country’s cultural solidarity and to drive real wages down—the question we asked in the title of the first article in this series is looking better and better. Has Obama really gone Bulworth on alien smuggling?

David Martin

October 12, 2014

See also “H-2A Kingpin Stumbles on H-2B.”

Addendum

 

“We do not like to be quarrelsome even though we are exceptional at it.”

That is a key passage in a 2008 intimidating email written by Lee Wicker to Charlene Giles of the Employment and Training Administration (ETA) of the U.S. Department of Labor, the federal agency that oversees the foreign guest worker program. Their quarrelsomeness will apparently soon be put to the test in in federal court.

David Martin

October 21, 2014

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Genuineness of Opinion

quotes-genuine-sounds-howard-thurman-480x480

Lately I have had several conversations with people I know in person (and those that I meet here in Cyber Land) where the subject of TROOFERS (Truthers) came up. Basically, what I look for are people who are genuine. I can have enlightening conversations and learn quite a bit from people I feel are honest and mean well. Where I have a problem, especially on line, is with those who I begin a dialogue with and find that they are full of shit, or worse, intentionally trying to lead people astray.

Its not simply a personality conflict, but a gut level instinct that tells me I should not trust that person. Maybe its a small lie. Maybe its just jumping in on a meme and too bull-headed to turn away when proved wrong. Maybe they are paid to tell bullshit. Who knows? I just know that something happens where I become convinced that the person is not wholly trustworthy. Not genuine.

It doesn’t matter to me if I disagree with someone, as long as we can discuss the differences and I feel that the person is honestly portraying 28cbdec938d6e2ef3e3ddf50260e8889what they believe. In person, I have virtually no problem being able to have a conversation and judge fairly readily if they are full of shit or not. We have so many non-verbal clues that are unavailable in this cyber world.

The one thing I hope that people who visit here feel is that (right or wrong) I am genuine about my opinion (this is an opinion blog and a place where I share other REAL writers work like DC Dave). With Dave, I feel he is genuine and has a motive to help people understand the world we live in is full of lies and misdirections to keep us baffled and in the dark. If I didn’t, I sure as hell wouldn’t put up his work. It is the same with Kenny, Greg Bacon, Digger, Noor, Whitewraithe and most of these blogs that you see linked on the right hand side (some are admittedly informational and I don’t personally discuss much with them). It is the same with Anthony (who comments frequently), even though I may not always agree 100% (or even understand) some of what he shares. Same with almost everyone who comments here (and I do appreciate your contribution very much).

I probably don’t say that near enough.

There are many others that I have shared their work in the past and found out after the fact (as I learned more) that they are, in fact, liars, set to deceive for some motive. At first, when I became suspicious, I would try to discuss the issue with key questions (usually knowing the answer before it was given), testing them. People whom would offer good information most of the time would say or write something very odd, which caused me to question their motive and I would get one of my gut feelings (instinct, I suppose) telling me there is something amiss.

genuine_fry_futurama2It happened early on with a guy who once participated here (named Glenn). He turned into a weird fruitcake and began to attack me here over things that were meaningless. It happened with Kelso (who contributed here for a while) until his true Jewish self started to shine. Rosie is genuine, but I felt her separate as I became more Jew-wise.

And its the same with many other vaunted TROOFERS. I don’t think these people are honest or genuine in their actions. I feel there is an ulterior motive to their work. I once linked to their work, but soon took them all down because they could not be trusted, in my opinion.

The problem is that I know there are mutual folks who share my stuff and participate here that may not agree with my opinion on those that they still saddle up with. They may feel that these folks are giving them the straight poop (just like I did at one time). But that doesn’t mean I think they are full of shit because they may not have learned the same lesson I did (yet).

And here is the rub.

On many issues, I will be in agreement with those people. But that is how disinformation works, 95% truth and 5% of Bullshit. And its the bullshit that makes the other 95% suspect.

The only cross word Kenny and I ever had was over a moron that kept posting bullshit here, then at Kenny’s place (bringing Kenny’s aDW-no-one-can-really-tell-and-no-one-really-caresSideshow in on the outrageous behavior he was showing here). I took the bastard to task, spanked him with a good cyber back hand and Kenny wrote me to ask me if I minded him deleting my comment (of course, I did not mind).

That was the nature of Kenny. Slow to anger, quick to reach out and mend. He was genuine. The guy he was protecting is/was not. That is why he is no longer allowed to comment here, because virtually everything he writes is offensive and offers nothing to the dialogue.

I am not wasting my time with dick heads. There are but around 4 or 5 people that I have ever banned here, always because they are Richard Noggins and generally because they attack another commenter without any apologies.

The links you see on the right hand side have changed somewhat over the years as I progressed in knowledge. But there are a core of bloggers that have never changed (including Kenny, Greg, Noor and a few others). I think they are genuine, even though I don’t always agree with their sources. But I think they want to share information and allow us to make up our own minds.

pic Genuine 2

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BuelaHuh?

Did I rub you the wrong way or stroke you just right? Let me know below in the comments section or Email me at buelahman {AT} g m a i l {DOT} com

If for some reason you actually liked this post, click the “Like” button below. If you feel like someone else needs to see this (or you just want to ruin someone’s day), click the Share Button at the bottom of the post and heap this upon some undeserving soul. And as sad as this thought may be, it may be remotely possible that us rednecks here at The Revolt please you enough (or more than likely, you are just a glutton for punishment??), that you feel an overwhelming desire to subscribe via the Email subscription and/or RSS Feed buttons found on the upper right hand corner of this page (may the Lord have mercy on your soul).

All posts are opinions meant to foster comment, reporting, teaching & study under the “fair use doctrine” in Sec. 107 of U.S. Code Title 17. No statement of fact is made or should be implied. Ads appearing on this blog are solely the product of the advertiser and do not necessarily reflect the opinions of BuehlahMan’s Revolt or WordPress.com

RIP Kenny

One of the most insightful, gracious, and important influences on me as a blogger (and an online friend), Kenny (of Kenny’s Sideshow) has passed away. He was an inspiration to me and many others. He had a knack for being levelheaded and could research subject matter with the best of them. He had an excellent taste in music and featured hardly-known golden nuggets often. He was inventive and funny. He was one of my first links when I started this blog and have featured his posts/wisdom here many times (including making this video and this video from his imaginative side).

He passed away suddenly on Sept 18. He was 62 years old.

Although I never met him in person (having only spoken with him on the phone a few times, had many email exchanges, and the sharing of comments on each others blog), I considered him a friend.

From his obituary (I did confirm the email notification):

He was a wonderful man who loved his family more than anything.  He also loved his fur children, his Jack Russells, Taz and Tessa.  Kenneth enjoyed music and played guitar, mandolin and drums.

He will be missed.

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BuelaHuh?

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All posts are opinions meant to foster comment, reporting, teaching & study under the “fair use doctrine” in Sec. 107 of U.S. Code Title 17. No statement of fact is made or should be implied. Ads appearing on this blog are solely the product of the advertiser and do not necessarily reflect the opinions of BuehlahMan’s Revolt or WordPress.com

Cannabis Cures Cancer by Dr Sircus

Cannabis Cures Cancer

by Dr Sircus

marijuana2_clip_image011

There should be no more confusion about whether or not marijuana is effective for cancer patients. Medical marijuana is chemotherapy, natural style, for all cancer patients. The two forms of hemp oil, one with THC and CBD and the other CBD alone (which is pretty much legal everywhere) provide the body with chemo therapeutics without the danger and staggering side effects. There are many chapters in this book about cancer patients using marijuana but in this one we present a quick overview of the science that backs up the assertion that every cancer patient and every oncologist should put medical marijuana on their treatment maps.

What you will see in this chapter is reference to many scientific studies that are all viewable on governmental sites. The United States government is pathetic in its dishonesty about medical marijuana both believing in it and holding patents for its medical use and claiming at the same time that it has no medical use. The federal government and still many states would rather throw innocent people in jail for using medical marijuana than be honest about how much it can help people recover from cancer and other diseases.

Below are summaries to just some of the scientific research out there that sustains the belief that medical marijuana will help people cure their cancer.

One of the most exciting areas of current research in the cannabinoid field is the study of the potential application of these compounds as antitumor drugs. CBD represents the first nontoxic exogenous agent that can significantly decrease Id-1 expression in metastatic breast cancer cells leading to the down-regulation of tumor aggressiveness.[1],[2] The CBD concentrations effective at inhibiting Id-1 expression correlated with those used to inhibit the proliferative and invasive phenotype of breast cancer cells. Of the five cannabinoids tested: cannabidiol, cannabigerol, cannnabichromene; cannabidiol-acid and THC-acid, it was found that cannabidiol is the most potent inhibitor of cancer cell growth. Taken together, these data might set the bases for a cannabinoid therapy for the management of breast cancer.[3]

marijuana2_clip_image012

Results show that Δ9-tetrahydrocannabinol reduces tumor growth, tumor number, and the amount/severity of lung metastases in MMTV-neu mice.[4] Cannabinoids induce ICAM-1, thereby conferring TIMP-1 induction and subsequent decreased cancer cell invasiveness thus inhibits lung cancer invasion and metastasis.[5]

Non-small cell lung cancer (NSCLC) is the leading cause of cancer deaths worldwide. Researchers have observed expression of CB1 (24%) and CB2 (55%) in NSCLC patients. They have also shown that the treatment of NSCLC cell lines (A549 and SW-1573) with CB1/CB2- and CB2-specific agonists Win55,212-2 and JWH-015, respectively, significantly attenuated random as well as growth factor-directed in vitro chemotaxis and chemoinvasion in these cells.[6]

Researchers in lung cancers also reported that they observed significant reduction in focal adhesion complex, which plays an important role in cancer migration. Medical marijuana significantly inhibited in vivo tumor growth and lung metastasis (∼50%).[7]

In research on pancreatic cancer it was found that cannabinoids lead to apoptosis of pancreatic tumor cells via a CB2 receptor and de novo synthesized ceramide-dependent up-regulation of p8 and the endoplasmic reticulum stress–related genes ATF-4 and TRB3. These findings may contribute to set the basis for a new therapeutic approach for the treatment of pancreatic cancer as reported by the National Cancer Institute.

Prostate cancer cells possess increased expression of both cannabinoid 1 and 2 receptors, and stimulation of these results in decrease in cell viability, increased apoptosis, and decreased androgen receptor expression and prostate-specific antigen excretion.[8]

In colorectal carcinoma cell lines, cannabidiol protected DNA from oxidative damage, increased endocannabinoid levels and reduced cell proliferation in a CB(1)-, TRPV1- and PPARγ-antagonists sensitive manner. It is concluded that cannabidiol exerts chemopreventive effect in vivo and reduces cell proliferation through multiple mechanisms.[9]

Ovarian cancer represents one of the leading cause of cancer-related deaths for women and is the most common gynecologic malignancy. Results with medical marijuana support a new therapeutic approach for the treatment of ovarian cancer. It is also conceivable that with available cannabinoids as lead compounds, non-habit forming agents that have higher biological effects could be developed.[10]

Examination of a number of human leukaemia and lymphoma cell lines demonstrate that CB2 cannabinoid receptors expressed on malignancies of the immune system may serve as potential targets for the induction of apoptosis. Also, because CB2 agonists lack psychotropic effects, they may serve as novel anticancer agents to selectively target and kill tumors of immune origin.[11] Plant-derived cannabinoids, including Delta9-tetrahydrocannabinol (THC), induce apoptosis in leukemic cells.[12]

Cannabinoid-treated tumors showed an increased number of apoptotic cells. This was accompanied by impairment of tumor vascularization, as determined by altered blood vessel morphology and decreased expression of proangiogenic factors (VEGF, placental growth factor, and angiopoietin. Abrogation of EGF-R function was also observed in cannabinoid-treated tumors.[13] These results support a new therapeutic approach for the treatment of skin tumors.

Hepatocellular carcinoma (HCC) is the third cause of cancer-related death worldwide. When these tumors are in advanced stages, few therapeutic options are available. In this study, the effects of cannabinoids–a novel family of potential anticancer agents–on the growth of HCC was investigated. It was found that Δ(9)-tetrahydrocannabinol (Δ(9)-THC, the main active component of Cannabis sativa) and JWH-015 (a cannabinoid receptor 2 (CB(2)) cannabinoid receptor-selective agonist) reduced the viability of the human HCC cell lines Cannabinoids were able to inhibit tumor growth and ascites in an orthotopic model of HCC xenograft.[14] These findings may contribute to the design of new therapeutic strategies for the management of HCC.

Both cholangiocarcinoma cell lines and surgical specimens from cholangiocarcinoma patients expressed cannabinoid receptors. THC inhibited cell proliferation, migration and invasion, and induced cell apoptosis. THC also decreased actin polymerization and reduced tumor cell survival in anoikis assay. pMEK1/2 and pAkt demonstrated the lower extent than untreated cells. Consequently, THC is potentially used to retard cholangiocarcinoma cell growth and metastasis.[15]

Smoking marijuana might decrease the smoker’s risk for bladder cancer, a new study shows. Retrospectively analyzing a large database of patients, researchers at Kaiser Permanente in California found that patients who reported cannabis use were 45% less likely to be diagnosed with bladder cancer than patients who did not smoke at all.

THC is a potent inducer of apoptosis, even at 1 x IC(50) (inhibitory concentration 50%) concentrations and as early as 6 hours after exposure to the drug. These effects were seen in leukemic cell lines (CEM, HEL-92, and HL60) as well as in peripheral blood mononuclear cells.[16] Cannabinoids represent a novel class of drugs active in increasing the life span in  mice carrying Lewis lung tumors and decreasing primary tumor size.[17]

marijuana2_clip_image014

Research has also found a cannabidiol-driven impaired invasion of human cervical cancer (HeLa, C33A) and human lung cancer cells (A549) that was reversed by antagonists to both CB(1) and CB(2) receptorrs as well as to transient receptor potential vanilloid 1 (TRPV1). The decrease of invasion by cannabidiol appeared concomitantly with up regulation of tissue inhibitor of matrix metalloproteinases-1 (TIMP the findings provide a novel mechanism underlying the anti-invasive action of cannabidiol and imply its use as a therapeutic option for the treatment of highly invasive cancers.[18]

A new anticancer quinone (HU-331) was synthesized from cannabidiol. It shows significant high efficacy against human cancer cell lines in vitro and against in vivo tumor grafts in nude mice. Two non-psychotropic cannabinoids, cannabidiol (CBD) and cannabidiol-dimethylheptyl (CBD-DMH), induced apoptosis in a human acute myeloid leukemia (AML) HL-60 cell line.[19]

Other studies show a synthetic and potent cannabinoid receptor agonist, investigated in hepatoma HepG2 cells and a possible signal transduction pathway that is proposed, indicates a potential positive role in liver cancer.[20] Cannabinoids have been found to counteract intestinal inflammation and colon cancer.[21]

The control of the cellular proliferation has become a focus of major attention as opening new therapeutic possibilities for the use of cannabinoids as potential antitumor agents.[22] Cannabinoid treatment inhibits angiogenesis of gliomas in vivo.[23] Remarkably, cannabinoids kill glioma cells selectively and can protect non-transformed glial cells from death. These and other findings reviewed here might set the basis for a potential use of cannabinoids in the management of gliomas. Other confirming studies may provide the basis for a new therapeutic approach for the treatment of malignant gliomas.[24]

In summary

Cannabinoids are found to exert their anti-cancer effects in a number of ways and in a variety of tissues.

  • Triggering cell death, through a mechanism called apoptosis
  • Stopping cells from dividing
  • Preventing new blood vessels from growing into tumours
  • Reducing the chances of cancer cells spreading through the body, by stopping cells from moving or invading neighbouring tissue
  • Speeding up the cell’s internal ‘waste disposal machine’ – a process known as autophagy – which can lead to cell death

All these effects are thought to be caused by cannabinoids locking onto the CB1 and CB2 cannabinoid receptors.  Almost daily we are seeing new or confirming evidence that Cannibinoids can be used to great benefit in cancer treatment of many types.

mm

Buy the new Medical Marijuana 2nd Edition eBook!

[1] Crosstalk between chemokine receptor CXCR4 and cannabinoid receptor CB2 in modulating breast cancer growth and invasion. Nasser MW; et al; PLoS One. 2011;6(9):e23901. doi: 10.1371/journal.pone.0023901. Epub 2011 Sep 7;  http://www.ncbi.nlm.nih.gov/pubmed/21915267 .

[2] Cannabidiol as a novel inhibitor of Id-1 gene expression in aggressive breast cancer cells; McAllister SDet al; Mol Cancer Ther. 2007 Nov;6(11):2921-7; http://www.ncbi.nlm.nih.gov/pubmed/18025276.

[3] Delta9-tetrahydrocannabinol inhibits cell cycle progression in human breast cancer cells through Cdc2 regulation; Caffarel MM et al; Cancer Res; 2006 Jul 1;66(13):6615-21; http://www.ncbi.nlm.nih.gov/pubmed/16818634

[4] Cannabinoids: a new hope for breast cancer therapy?
Caffarel MM et al; Cancer Treat Rev.: 2012 Nov; 38(7):911-8. doi: 10.1016/j.ctrv.2012.06.005. Epub 2012 Jul 7;  http://www.ncbi.nlm.nih.gov/pubmed/22776349

[5] Cannabidiol inhibits lung cancer cell invasion and metastasis via intercellular adhesion molecule-1.Ramer R et al; FASEB J.; 2012 Apr;26(4):1535-48. doi: 10.1096/fj.11-198184. Epub 2011 Dec 23; http://www.ncbi.nlm.nih.gov/pubmed/22198381?dopt=Abstract

[6] Cannabinoid receptors, CB1 and CB2, as novel targets for inhibition of non-small cell lung cancer growth and metastasis; Preet A, et al; Cancer Prev Res (Phila). 2011 Jan;4(1):65-75. doi: 10.1158/1940-6207.CAPR-10-0181. Epub 2010 Nov 19; http://www.ncbi.nlm.nih.gov/pubmed/21097714?dopt=Abstract

[7] Δ9-Tetrahydrocannabinol inhibits epithelial growth factor-induced lung cancer cell migration in vitro as well as its growth and metastasis in vivo; A Preetet al; Oncogene; (2008) 27, 339–346; doi:10.1038/sj.onc.1210641; published online 9 July 2007;  http://www.nature.com/onc/journal/v27/n3/abs/1210641a.html

[8] The role of cannabinoids in prostate cancer: Basic science perspective and potential clinical applications;Juan A. Ramos and Fernando J. Bianco; Indian J Urol. 2012 Jan-Mar; 28(1): 9–14;.doi:10.4103/0970-1591.94942http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3339795/?report=classic

[9] Chemopreventive effect of the non-psychotropic phytocannabinoid cannabidiol on experimental colon cancer.
Aviello G et al; ;J Mol Med (Berl);2012 Aug;90(8):925-34. doi: 10.1007/s00109-011-0856-x. Epub 2012;  Jan 10.;  http://www.ncbi.nlm.nih.gov/pubmed/22231745

[10] Cannabinoid receptors as a target for therapy of ovarian cancer

Farrukh Afaq; et al;,Proc Amer Assoc Cancer Res, Volume 47, 2006; http://www.aacrmeetingabstracts.org/cgi/content/abstract/2006/1/1084

[11] Targeting CB2 cannabinoid receptors as a novel therapy to treat malignant lymphoblastic disease.
McKallip RJ et al; Blood. 2002 Jul 15;100(2):627-34.; http://www.ncbi.nlm.nih.gov/pubmed/12091357

[12] Delta9-tetrahydrocannabinol-induced apoptosis in Jurkat leukemia T cells is regulated by translocation of Bad to mitochondria.
Jia W et al; Mol Cancer Res.; 2006 Aug;4(8):549-62; http://www.ncbi.nlm.nih.gov/pubmed/16908594 .

[13] Inhibition of skin tumor growth and angiogenesis in vivo by activation of cannabinoid receptors.
Casanova ML et al: J Clin Invest. 2003 Jan;111(1):43-50; http://www.ncbi.nlm.nih.gov/pubmed/12511587

[14] Anti-tumoral action of cannabinoids on hepatocellular carcinoma: role of AMPK-dependent activation of autophagy. Vara D et al; Cell Death Differ; 2011 Jul;18(7):1099-111. doi: 10.1038/cdd.2011.32. Epub 2011 Apr 8.; http://www.ncbi.nlm.nih.gov/pubmed/21475304

[15] The dual effects of delta(9)-tetrahydrocannabinol on cholangiocarcinoma cells: anti-invasion activity at low concentration and apoptosis induction at high concentration. Leelawat Set al; Cancer Invest. 2010 May;28(4):357-63. doi: 10.3109/07357900903405934; http://www.ncbi.nlm.nih.gov/pubmed/19916793.

[16] Cannabis-induced cytotoxicity in leukemic cell lines: the role of the cannabinoid receptors and the MAPK pathway;Powles T et al; Blood;.2005 Feb 1;105(3):1214-21; Epub 2004 Sep 28.; http://www.ncbi.nlm.nih.gov/pubmed/15454482

[17] In vivo effects of cannabinoids on macromolecular biosynthesis in Lewis lung carcinomas; Friedman MA; Cancer Biochem Biophys. 1977;2(2):51-4.; http://www.ncbi.nlm.nih.gov/pubmed/616322

[18] Cannabidiol inhibits cancer cell invasion via upregulation of tissue inhibitor of matrix metalloproteinases-1; Ramer Ret al; Biochem Pharmacol; 2010 Apr 1;79(7):955-66. doi: 10.1016/j.bcp.2009.11.007. Epub 2009 Nov 13; http://www.ncbi.nlm.nih.gov/pubmed/19914218

[19] Gamma-irradiation enhances apoptosis induced by cannabidiol, a non-psychotropic cannabinoid, in cultured HL-60 myeloblastic leukemia cells. Gallily R et al; Leuk Lymphoma.: 2003 Oct;44(10):1767-73; http://www.ncbi.nlm.nih.gov/pubmed/14692532.

[20] Apoptosis induced in HepG2 cells by the synthetic cannabinoid WIN: involvement of the transcription factor PPARgamma. Giuliano Met et al; Biochimie;. 2009 Apr;91(4):457-65. doi: 10.1016/j.biochi.2008.11.003. Epub 2008 Nov 27. http://www.ncbi.nlm.nih.gov/pubmed/19059457

[21] Cannabinoids in intestinal inflammation and cancer. Izzo AA1, Camilleri M.; Pharmacol Res; 2009 Aug;60(2):117-25. doi: 10.1016/j.phrs.2009.03.008. Epub 2009 Mar 18; http://www.ncbi.nlm.nih.gov/pubmed/19442536

[22] Involvement of cannabinoids in cellular proliferation;López-Rodríguez ML et al; ;Mini Rev Med Chem; 2005 Jan;5(1):97-106 http://www.ncbi.nlm.nih.gov/pubmed/15638794

[23] Hypothesis: cannabinoid therapy for the treatment of gliomas? Velasco G et al;  Neuropharmacology;.2004 Sep;47(3):315-23; http://www.ncbi.nlm.nih.gov/pubmed/15275820

[24] Anti-tumoral action of cannabinoids: involvement of sustained ceramide accumulation and extracellular signal-regulated kinase activation; Galve-Roperh; Nat Med.; 2000 Mar;6(3):313-9; http://www.ncbi.nlm.nih.gov/pubmed/10700234

 


 

h/t GreenMedInfo

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The Jewish Takeover of Canada: The Case of Arthur Topham

The Jewish Takeover of Canada: The Case of Arthur Topham

by

Has Canada been taken over by Jews? It would seem so, if the relentless persecution of Canadian patriot and freedom fighter Arthur Topham is anything to go on.

jewtakecan

IT’S TOO LATE TO CRY — IT’S ALREADY HAPPENED

Canadian patriot and freedom fighter Arthur Topham is to be hauled before the Canadian courts next year on trumped-up charges. There is absolutely no doubt in my mind that Arthur is an innocent man. His trial date has now been set. He will appear in court on 26 October, 2015, and his trial will last for two weeks until 6 November.

If found guilty, this man who has said and done nothing that you and I have not said or done a thousand times, will be torn from the embrace of his wife, his family and his friends and be thrown into prison. It will be a major miscarriage of justice if this should occur. 

The forthcoming trial of Arthur Topham is therefore much more than the trial of one man. Canadian justice will itself be on trial.

arthur tophamWhat is Arthur’s alleged crime? Arthur’s only crime is that he is a political dissident who has chosen to exercise his democratic right to free speech. He has spoken out eloquently about the war crimes of the state of Israel and published books on his website which are regarded as offensive to many Jews. To criticize this privileged ethnic group in Canada or to question its cherished assumptions, is, it seems, strictly taboo. This is classified as “hate speech”.

At no time has Arthur advocated breaking the law. He has never incited anyone to violence. He has merely utilized his pen to express his political views in a rational and civilized way as any political dissident anywhere in the world would do.

If Arthur had been an American or British citizen, he would not be facing a possible prison sentence right now, and this is because whatever Arthur has said or done is not regarded as a crime in the United States or Britain.

If Arthur is to be condemned in a Canadian court for “hate speech”, it will only be because Canada has now fallen under the dominant influence of a powerful ethnic group who have somehow managed to turn Canada into an Israelified police state.

 

Please read the rest at Darkmoon’s site:

http://www.darkmoon.me/2014/defense-arthur-topham/

 

B’Man: Good luck, Mr Topham. This entire thing is a blatant sham. I hope the very best for you and your family as you beat it.

 

Putin Never Quits


DC Dave’s parody derived from Puttin’ On The Ritz

Did you hear the Kagan wife and her recipe for strife
With the crass effrontery to push U.S. hegemony?
A hacker heard the callers making plans for using dollars
For neocon reign, this time in Ukraine.
If you’re blue and need it spelled out for you
It’s called the doctrine Wolfowitz…but hey,
Putin never quits.

Provocateur snipers, and mercenary vipers,
We all know it’s the pits,
But Putin never quits.

Prescribing a one-size-fits-all solution
And calling it color revolution (pollution)
Make them mix where global bankers swing big sticks,
We’re taking off the mitts,
But Putin never quits.

Now I’d like to ask you Daddyo
Have you heard of Operation Gladio? Oh no?
It’s NATO’s very rotten underbelly,
But what’s giving us fits?
Putin never quits.

Original poem:

http://dcdave.com/poet15/140606.htm

Video and singing by BuelahMan
http://www.buelahman.wordpress.com

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All posts are opinions meant to foster comment, reporting, teaching & study under the “fair use doctrine” in Sec. 107 of U.S. Code Title 17. No statement of fact is made or should be implied. Ads appearing on this blog are solely the product of the advertiser and do not necessarily reflect the opinions of BuehlahMan’s Revolt or WordPress.com