Another Weed Lie Abolished: Smoking Does NOT Make You Stupid

Even in children:

Latest science shows there is no relationship between cannabis use and lower IQ

From Alternet

Moderate cannabis consumption by young people is not positively associated with changes in intelligence quotient (IQ), according to data [2] presented this week at the European College of Neuropsychopharmacology annual congress in Berlin, Germany.

Investigators at the University College of London analyzed data from 2,612 subjects who had their IQ tested at the age of eight and again at age 15. They reported no relationship between cannabis use and lower IQ at age 15 [3] when confounding factors such as subjects’ history of alcohol use and cigarette use were taken into account.

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“In particular alcohol use was found to be strongly associated with IQ decline,” the authors wrote in a press release cited [3] by The Washington Post. “No other factors were found to be predictive of IQ change.”

Quoted in the Independent Business Times, the study’s lead author said: “Our findings suggest cannabis may not have a detrimental effect on cognition, once we account for other related factors particularly cigarette and alcohol use. This may suggest that previous research findings showing poorer cognitive performance in cannabis users may have resulted from the lifestyle, behavior and personal history typically associated with cannabis use, rather than cannabis use itself.”

The investigators acknowledged that more chronic marijuana use, defined in the study as a subject’s admission of having consumed cannabis 50 times or more by age 15, was correlated with slightly poorer exam results at the age of 16 — even after controlling for other variables. However, investigators admitted [2]: “It’s hard to know what causes what. Do kids do badly at school because they are smoking weed, or do they smoke weed because they’re doing badly?”

Commenting on the newly presented data, the meeting’s Chair, Guy Goodwin, from the University of Oxford, told [2] BBC News: “This is a potentially important study because it suggests that the current focus on the alleged harms of cannabis may be obscuring the fact that its use is often correlated with that of other even more freely available drugs and possibly lifestyle factors.”

In a recent review published in the New England Journal of Medicine, the NIDA Director Nora Volkow alleged [4] that cannabis use, particularly by adolescents, is associated with brain alterations and lower IQ. However, the IQ study [5] cited by Ms. Volkow as the basis of her claim was later questioned in a separate analysis published in the Proceedings of the National Academy of Sciences. That paper suggested [6] that socioeconomics, not subjects’ cannabis use, was responsible for differences in IQ and that the plant’s “true effect [on intelligence quotient] could be zero.”

previous assessment [7] of cannabis use and its potential impact on intelligence quotient in a cohort of young people tracked since birth reported, “[M]arijuana does not have a long-term negative impact on global intelligence.”

MARCH 15. 2014 (20)

pt_1022_345_oWe were permeated with lies as we grew up. I came up in the late 60’s and 70’s when it was more accepted, but still had the stigma (it was just the beginning of the Drug War and everyone knew Nixon defied his own advisers to do so). The Drug War Profiteers have used every imaginable lie and distortion to keep this medicine from your hands, especially the tired old “it’s for the children” garbage. Even many commenters here over the years have argued with me on this very subject, when I knew anecdotally and via experience with many teens over the years that it was mostly scare tactics, perpetuating the lies to keep the MEDICINE illegal (thereby keeping Pharmacuetical companies, drug rehabs, private and state prisons in business; not to mention the extra loot for the police force that would rather do the easy job of busting the stink instead of, say, busting rapers.

According to the Department of Justice, there are currently over 400,000 untested rape kits collecting dust in police evidence rooms nationwide, and many other estimates suggest that this number could be as high as one million.

As a result of this horrific negligence, roughly 3% of rape cases in America are actually solved, despite the fact that many rape kits have a high chance of leading to an arrest, since most rapists are career criminals who have their DNA on file.

In some cases, the victims even know who their attackers were, but they can not prosecute these criminals because the evidence has not been processed by police.

Bobbie Villareal, Executive Director of the Dallas Area Rape Crisis Center, admitted that many police departments cut corners in rape cases because testing is expensive, and the investigation requires a lot of work.

Busting for the stink doesn’t take much work.

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Now, just because I call out the liars on this subject does not mean that I think children should be getting high. I don’t. I won’t want my child doing it until she is grown and makes that decision for herself. So, why the seeming contradiction? Because the one thing I know weed CAN do is demotivate certain personality types (and that has a lot to do with the type and potency of the cannabis). I have known people who smoked the joints too much and ended up on the couch with no motivation to do much of anything.

As a matter of fact, one of the smartest people I know was a very heavy pot head. He would wake and bake and smoke all day long. He didn’t want to do much else. (On the other hand, I worked with a guy in construction that was high all day and could out work anyone on the site)

picture-1-67-tmTHIS, if anything, should be the warning to kids (to anyone, for that matter). Know what you are partaking of and how it affects you. Limit your usage if you do not want to abstain. Find people with experience that you can talk to (but be careful of the narcs ready to snitch on anyone to get that blue pat on the back). Best thing is to wait until you are an adult and move to where it is legal. Someday, I believe in the next few short years, it will legalized virtually everywhere in the US, except for the most backwards states such as mine.

Realistically, though, if this substance was legal and readily available like alcohol, people would learn which strain helps them the most without the lethargy and demotivation.

As it is, you take what you can get and hope for the best. Legalized, all of that goes away and you choose exactly which “bourbon” or “Scotch”, “Irish whiskey” you desire and works for you.

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h/t Patients4MedicalMarijuana and The Free Thought Project

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Cannabis Cures Cancer by Dr Sircus

Cannabis Cures Cancer

by Dr Sircus

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There should be no more confusion about whether or not marijuana is effective for cancer patients. Medical marijuana is chemotherapy, natural style, for all cancer patients. The two forms of hemp oil, one with THC and CBD and the other CBD alone (which is pretty much legal everywhere) provide the body with chemo therapeutics without the danger and staggering side effects. There are many chapters in this book about cancer patients using marijuana but in this one we present a quick overview of the science that backs up the assertion that every cancer patient and every oncologist should put medical marijuana on their treatment maps.

What you will see in this chapter is reference to many scientific studies that are all viewable on governmental sites. The United States government is pathetic in its dishonesty about medical marijuana both believing in it and holding patents for its medical use and claiming at the same time that it has no medical use. The federal government and still many states would rather throw innocent people in jail for using medical marijuana than be honest about how much it can help people recover from cancer and other diseases.

Below are summaries to just some of the scientific research out there that sustains the belief that medical marijuana will help people cure their cancer.

One of the most exciting areas of current research in the cannabinoid field is the study of the potential application of these compounds as antitumor drugs. CBD represents the first nontoxic exogenous agent that can significantly decrease Id-1 expression in metastatic breast cancer cells leading to the down-regulation of tumor aggressiveness.[1],[2] The CBD concentrations effective at inhibiting Id-1 expression correlated with those used to inhibit the proliferative and invasive phenotype of breast cancer cells. Of the five cannabinoids tested: cannabidiol, cannabigerol, cannnabichromene; cannabidiol-acid and THC-acid, it was found that cannabidiol is the most potent inhibitor of cancer cell growth. Taken together, these data might set the bases for a cannabinoid therapy for the management of breast cancer.[3]

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Results show that Δ9-tetrahydrocannabinol reduces tumor growth, tumor number, and the amount/severity of lung metastases in MMTV-neu mice.[4] Cannabinoids induce ICAM-1, thereby conferring TIMP-1 induction and subsequent decreased cancer cell invasiveness thus inhibits lung cancer invasion and metastasis.[5]

Non-small cell lung cancer (NSCLC) is the leading cause of cancer deaths worldwide. Researchers have observed expression of CB1 (24%) and CB2 (55%) in NSCLC patients. They have also shown that the treatment of NSCLC cell lines (A549 and SW-1573) with CB1/CB2- and CB2-specific agonists Win55,212-2 and JWH-015, respectively, significantly attenuated random as well as growth factor-directed in vitro chemotaxis and chemoinvasion in these cells.[6]

Researchers in lung cancers also reported that they observed significant reduction in focal adhesion complex, which plays an important role in cancer migration. Medical marijuana significantly inhibited in vivo tumor growth and lung metastasis (∼50%).[7]

In research on pancreatic cancer it was found that cannabinoids lead to apoptosis of pancreatic tumor cells via a CB2 receptor and de novo synthesized ceramide-dependent up-regulation of p8 and the endoplasmic reticulum stress–related genes ATF-4 and TRB3. These findings may contribute to set the basis for a new therapeutic approach for the treatment of pancreatic cancer as reported by the National Cancer Institute.

Prostate cancer cells possess increased expression of both cannabinoid 1 and 2 receptors, and stimulation of these results in decrease in cell viability, increased apoptosis, and decreased androgen receptor expression and prostate-specific antigen excretion.[8]

In colorectal carcinoma cell lines, cannabidiol protected DNA from oxidative damage, increased endocannabinoid levels and reduced cell proliferation in a CB(1)-, TRPV1- and PPARγ-antagonists sensitive manner. It is concluded that cannabidiol exerts chemopreventive effect in vivo and reduces cell proliferation through multiple mechanisms.[9]

Ovarian cancer represents one of the leading cause of cancer-related deaths for women and is the most common gynecologic malignancy. Results with medical marijuana support a new therapeutic approach for the treatment of ovarian cancer. It is also conceivable that with available cannabinoids as lead compounds, non-habit forming agents that have higher biological effects could be developed.[10]

Examination of a number of human leukaemia and lymphoma cell lines demonstrate that CB2 cannabinoid receptors expressed on malignancies of the immune system may serve as potential targets for the induction of apoptosis. Also, because CB2 agonists lack psychotropic effects, they may serve as novel anticancer agents to selectively target and kill tumors of immune origin.[11] Plant-derived cannabinoids, including Delta9-tetrahydrocannabinol (THC), induce apoptosis in leukemic cells.[12]

Cannabinoid-treated tumors showed an increased number of apoptotic cells. This was accompanied by impairment of tumor vascularization, as determined by altered blood vessel morphology and decreased expression of proangiogenic factors (VEGF, placental growth factor, and angiopoietin. Abrogation of EGF-R function was also observed in cannabinoid-treated tumors.[13] These results support a new therapeutic approach for the treatment of skin tumors.

Hepatocellular carcinoma (HCC) is the third cause of cancer-related death worldwide. When these tumors are in advanced stages, few therapeutic options are available. In this study, the effects of cannabinoids–a novel family of potential anticancer agents–on the growth of HCC was investigated. It was found that Δ(9)-tetrahydrocannabinol (Δ(9)-THC, the main active component of Cannabis sativa) and JWH-015 (a cannabinoid receptor 2 (CB(2)) cannabinoid receptor-selective agonist) reduced the viability of the human HCC cell lines Cannabinoids were able to inhibit tumor growth and ascites in an orthotopic model of HCC xenograft.[14] These findings may contribute to the design of new therapeutic strategies for the management of HCC.

Both cholangiocarcinoma cell lines and surgical specimens from cholangiocarcinoma patients expressed cannabinoid receptors. THC inhibited cell proliferation, migration and invasion, and induced cell apoptosis. THC also decreased actin polymerization and reduced tumor cell survival in anoikis assay. pMEK1/2 and pAkt demonstrated the lower extent than untreated cells. Consequently, THC is potentially used to retard cholangiocarcinoma cell growth and metastasis.[15]

Smoking marijuana might decrease the smoker’s risk for bladder cancer, a new study shows. Retrospectively analyzing a large database of patients, researchers at Kaiser Permanente in California found that patients who reported cannabis use were 45% less likely to be diagnosed with bladder cancer than patients who did not smoke at all.

THC is a potent inducer of apoptosis, even at 1 x IC(50) (inhibitory concentration 50%) concentrations and as early as 6 hours after exposure to the drug. These effects were seen in leukemic cell lines (CEM, HEL-92, and HL60) as well as in peripheral blood mononuclear cells.[16] Cannabinoids represent a novel class of drugs active in increasing the life span in  mice carrying Lewis lung tumors and decreasing primary tumor size.[17]

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Research has also found a cannabidiol-driven impaired invasion of human cervical cancer (HeLa, C33A) and human lung cancer cells (A549) that was reversed by antagonists to both CB(1) and CB(2) receptorrs as well as to transient receptor potential vanilloid 1 (TRPV1). The decrease of invasion by cannabidiol appeared concomitantly with up regulation of tissue inhibitor of matrix metalloproteinases-1 (TIMP the findings provide a novel mechanism underlying the anti-invasive action of cannabidiol and imply its use as a therapeutic option for the treatment of highly invasive cancers.[18]

A new anticancer quinone (HU-331) was synthesized from cannabidiol. It shows significant high efficacy against human cancer cell lines in vitro and against in vivo tumor grafts in nude mice. Two non-psychotropic cannabinoids, cannabidiol (CBD) and cannabidiol-dimethylheptyl (CBD-DMH), induced apoptosis in a human acute myeloid leukemia (AML) HL-60 cell line.[19]

Other studies show a synthetic and potent cannabinoid receptor agonist, investigated in hepatoma HepG2 cells and a possible signal transduction pathway that is proposed, indicates a potential positive role in liver cancer.[20] Cannabinoids have been found to counteract intestinal inflammation and colon cancer.[21]

The control of the cellular proliferation has become a focus of major attention as opening new therapeutic possibilities for the use of cannabinoids as potential antitumor agents.[22] Cannabinoid treatment inhibits angiogenesis of gliomas in vivo.[23] Remarkably, cannabinoids kill glioma cells selectively and can protect non-transformed glial cells from death. These and other findings reviewed here might set the basis for a potential use of cannabinoids in the management of gliomas. Other confirming studies may provide the basis for a new therapeutic approach for the treatment of malignant gliomas.[24]

In summary

Cannabinoids are found to exert their anti-cancer effects in a number of ways and in a variety of tissues.

  • Triggering cell death, through a mechanism called apoptosis
  • Stopping cells from dividing
  • Preventing new blood vessels from growing into tumours
  • Reducing the chances of cancer cells spreading through the body, by stopping cells from moving or invading neighbouring tissue
  • Speeding up the cell’s internal ‘waste disposal machine’ – a process known as autophagy – which can lead to cell death

All these effects are thought to be caused by cannabinoids locking onto the CB1 and CB2 cannabinoid receptors.  Almost daily we are seeing new or confirming evidence that Cannibinoids can be used to great benefit in cancer treatment of many types.

mm

Buy the new Medical Marijuana 2nd Edition eBook!

[1] Crosstalk between chemokine receptor CXCR4 and cannabinoid receptor CB2 in modulating breast cancer growth and invasion. Nasser MW; et al; PLoS One. 2011;6(9):e23901. doi: 10.1371/journal.pone.0023901. Epub 2011 Sep 7;  http://www.ncbi.nlm.nih.gov/pubmed/21915267 .

[2] Cannabidiol as a novel inhibitor of Id-1 gene expression in aggressive breast cancer cells; McAllister SDet al; Mol Cancer Ther. 2007 Nov;6(11):2921-7; http://www.ncbi.nlm.nih.gov/pubmed/18025276.

[3] Delta9-tetrahydrocannabinol inhibits cell cycle progression in human breast cancer cells through Cdc2 regulation; Caffarel MM et al; Cancer Res; 2006 Jul 1;66(13):6615-21; http://www.ncbi.nlm.nih.gov/pubmed/16818634

[4] Cannabinoids: a new hope for breast cancer therapy?
Caffarel MM et al; Cancer Treat Rev.: 2012 Nov; 38(7):911-8. doi: 10.1016/j.ctrv.2012.06.005. Epub 2012 Jul 7;  http://www.ncbi.nlm.nih.gov/pubmed/22776349

[5] Cannabidiol inhibits lung cancer cell invasion and metastasis via intercellular adhesion molecule-1.Ramer R et al; FASEB J.; 2012 Apr;26(4):1535-48. doi: 10.1096/fj.11-198184. Epub 2011 Dec 23; http://www.ncbi.nlm.nih.gov/pubmed/22198381?dopt=Abstract

[6] Cannabinoid receptors, CB1 and CB2, as novel targets for inhibition of non-small cell lung cancer growth and metastasis; Preet A, et al; Cancer Prev Res (Phila). 2011 Jan;4(1):65-75. doi: 10.1158/1940-6207.CAPR-10-0181. Epub 2010 Nov 19; http://www.ncbi.nlm.nih.gov/pubmed/21097714?dopt=Abstract

[7] Δ9-Tetrahydrocannabinol inhibits epithelial growth factor-induced lung cancer cell migration in vitro as well as its growth and metastasis in vivo; A Preetet al; Oncogene; (2008) 27, 339–346; doi:10.1038/sj.onc.1210641; published online 9 July 2007;  http://www.nature.com/onc/journal/v27/n3/abs/1210641a.html

[8] The role of cannabinoids in prostate cancer: Basic science perspective and potential clinical applications;Juan A. Ramos and Fernando J. Bianco; Indian J Urol. 2012 Jan-Mar; 28(1): 9–14;.doi:10.4103/0970-1591.94942http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3339795/?report=classic

[9] Chemopreventive effect of the non-psychotropic phytocannabinoid cannabidiol on experimental colon cancer.
Aviello G et al; ;J Mol Med (Berl);2012 Aug;90(8):925-34. doi: 10.1007/s00109-011-0856-x. Epub 2012;  Jan 10.;  http://www.ncbi.nlm.nih.gov/pubmed/22231745

[10] Cannabinoid receptors as a target for therapy of ovarian cancer

Farrukh Afaq; et al;,Proc Amer Assoc Cancer Res, Volume 47, 2006; http://www.aacrmeetingabstracts.org/cgi/content/abstract/2006/1/1084

[11] Targeting CB2 cannabinoid receptors as a novel therapy to treat malignant lymphoblastic disease.
McKallip RJ et al; Blood. 2002 Jul 15;100(2):627-34.; http://www.ncbi.nlm.nih.gov/pubmed/12091357

[12] Delta9-tetrahydrocannabinol-induced apoptosis in Jurkat leukemia T cells is regulated by translocation of Bad to mitochondria.
Jia W et al; Mol Cancer Res.; 2006 Aug;4(8):549-62; http://www.ncbi.nlm.nih.gov/pubmed/16908594 .

[13] Inhibition of skin tumor growth and angiogenesis in vivo by activation of cannabinoid receptors.
Casanova ML et al: J Clin Invest. 2003 Jan;111(1):43-50; http://www.ncbi.nlm.nih.gov/pubmed/12511587

[14] Anti-tumoral action of cannabinoids on hepatocellular carcinoma: role of AMPK-dependent activation of autophagy. Vara D et al; Cell Death Differ; 2011 Jul;18(7):1099-111. doi: 10.1038/cdd.2011.32. Epub 2011 Apr 8.; http://www.ncbi.nlm.nih.gov/pubmed/21475304

[15] The dual effects of delta(9)-tetrahydrocannabinol on cholangiocarcinoma cells: anti-invasion activity at low concentration and apoptosis induction at high concentration. Leelawat Set al; Cancer Invest. 2010 May;28(4):357-63. doi: 10.3109/07357900903405934; http://www.ncbi.nlm.nih.gov/pubmed/19916793.

[16] Cannabis-induced cytotoxicity in leukemic cell lines: the role of the cannabinoid receptors and the MAPK pathway;Powles T et al; Blood;.2005 Feb 1;105(3):1214-21; Epub 2004 Sep 28.; http://www.ncbi.nlm.nih.gov/pubmed/15454482

[17] In vivo effects of cannabinoids on macromolecular biosynthesis in Lewis lung carcinomas; Friedman MA; Cancer Biochem Biophys. 1977;2(2):51-4.; http://www.ncbi.nlm.nih.gov/pubmed/616322

[18] Cannabidiol inhibits cancer cell invasion via upregulation of tissue inhibitor of matrix metalloproteinases-1; Ramer Ret al; Biochem Pharmacol; 2010 Apr 1;79(7):955-66. doi: 10.1016/j.bcp.2009.11.007. Epub 2009 Nov 13; http://www.ncbi.nlm.nih.gov/pubmed/19914218

[19] Gamma-irradiation enhances apoptosis induced by cannabidiol, a non-psychotropic cannabinoid, in cultured HL-60 myeloblastic leukemia cells. Gallily R et al; Leuk Lymphoma.: 2003 Oct;44(10):1767-73; http://www.ncbi.nlm.nih.gov/pubmed/14692532.

[20] Apoptosis induced in HepG2 cells by the synthetic cannabinoid WIN: involvement of the transcription factor PPARgamma. Giuliano Met et al; Biochimie;. 2009 Apr;91(4):457-65. doi: 10.1016/j.biochi.2008.11.003. Epub 2008 Nov 27. http://www.ncbi.nlm.nih.gov/pubmed/19059457

[21] Cannabinoids in intestinal inflammation and cancer. Izzo AA1, Camilleri M.; Pharmacol Res; 2009 Aug;60(2):117-25. doi: 10.1016/j.phrs.2009.03.008. Epub 2009 Mar 18; http://www.ncbi.nlm.nih.gov/pubmed/19442536

[22] Involvement of cannabinoids in cellular proliferation;López-Rodríguez ML et al; ;Mini Rev Med Chem; 2005 Jan;5(1):97-106 http://www.ncbi.nlm.nih.gov/pubmed/15638794

[23] Hypothesis: cannabinoid therapy for the treatment of gliomas? Velasco G et al;  Neuropharmacology;.2004 Sep;47(3):315-23; http://www.ncbi.nlm.nih.gov/pubmed/15275820

[24] Anti-tumoral action of cannabinoids: involvement of sustained ceramide accumulation and extracellular signal-regulated kinase activation; Galve-Roperh; Nat Med.; 2000 Mar;6(3):313-9; http://www.ncbi.nlm.nih.gov/pubmed/10700234

 


 

h/t GreenMedInfo

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No, Weed Doesn’t Cause Lung Cancers

 

Study: Habitual Marijuana Smoking Not Associated With Increased Risk Of Lung Cancer

Thursday, 26 June 2014

Study: Habitual Marijuana Smoking Not Associated With Increased Risk Of Lung CancerToronto, Canada: Subjects who regularly inhale cannabis smoke possess no greater risk of contracting lung cancer than do those who consume it occasionally or not at all, according to data published online ahead of print in the International Journal of Cancer.

An international team of investigators from Canada, New Zealand, Great Britian, and the United States analyzed data from six case-control studies involving over 5,000 subjects (2,159 cases and 2,985 controls) from around the world.

Authors concluded, “Results from our pooled analyses provide little evidence for an increased risk of lung cancer among habitual or long-term cannabis smokers.”

The team had previously presented their data at the 2013 annual meeting of the American Academy for Cancer Research.

Their findings are similar to those of a 2013 review published in the journal Annals of the American Thoracic Society, which concluded: “[H]abitual use of marijuana alone does not appear to lead to significant abnormalities in lung function. … Overall, the risks of pulmonary complications of regular use of marijuana appear to be relatively small and far lower than those of tobacco smoking.”

An accompanying commentary appearing in the same journal affirmed, “[C]annabis smoking does not seem to increase risk of chronic obstructive pulmonary disease (COPD) or airway cancers. In fact, there is even a suggestion that at low doses cannabis may be protective for both conditions.”

Preclinical studies have documented that cannabinoids possess potent anti-cancer properties, including the inhibition of lung cancer cell growth. To date, however, scientists have yet to conduct controlled clinical trials replicating these results in human subjects.

For more information, please contact Paul Armentano, NORML Deputy Director, at: paul@norml.org. Full text of the study, “Cannabis smoking and lung cancer risk: Pooled analysis in the International Lung Cancer Consortium,” appears in the International Journal of Cancer.

 

To the little hater that gave me so much grief over this issue (the entire time I was explaining how she was incorrect and still regurgitating the government-spewed propaganda), take heed, learn truth and be woman enough to change your faulty logic and conclusions. Plainly put, you were/are wrong. Deal with it.

h/t NORML

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Vitamin Weed: The Vitamin Most Of Us Are Missing

So, what if one could get a bottle of liquid or capsules with cannabis oil vitamins within? What if the purpose was health related moreso than any buzz? What if humans were able to actually use the endocannabinoid system as it was intended to fight off the health related issues that are thrown upon us with environment, food and our lifestyles?


“Cannabis is the key to unlocking preventative medicine,” says neuroscientist Dr. Michelle Ross. “It helps protect your DNA from damage so it can actually slow down the aging process… I think everyone should use cannabis oil, you should learn about it, you shouldn’t be afraid.”

In this short clip from the ‘Reset with Amber Lyon’ podcast, neuroscientist Dr. Michele Ross explains why she believes everyone can benefit from daily use of cannabis oil, or ‘vitamin weed’ as she calls it. Dr. Ross is a former drug researcher for the National Institute on Drug Abuse, turned medical marijuana advocate.

To learn more about natural medicines and therapies please visit http://www.reset.me.

 Every day, pop a small capsule just like your Flintstones One-A-Day vitamins, except in this case, it prevents cancer from ever getting a foothold inside your body.

And by the way, anxiety causes many stresses and other health related problems. The fact that vitamin weed can actually help with that would go a long way into helping everyday Americans. And in that case, it can be smoked or vaporized.

Can you imagine a world where calm, thoughtful interaction ruled society?

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Evil, Wicked, Mean and Nasty

Even if you have heard this song before, you should really pay attention to the words. Look at the money wasted over lies. Lives ruined for ill-gotten profits.


Perhaps my favorite Weed Song.

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B’Man’s Sabbath Watch: Holy Shit!

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Rev. Edwin Sanders

The Rev. Edwin Sanders says churches should help heal the sick, feed the hungry and set prisoners free. Even if they smoke pot.

Dr. Forrest Harris

Dr Forrest Harris

“The war on drugs is a moral injustice.”

Rev.-John-Jackson-TUCC-Gary-cannabis-god

Rev John Jackson

“God does not care if you smoke weed. God is not that petty.”

 

I wanna know: Where da white folk at?

h/t USA Today

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BuelaHuh?

Did I rub you the wrong way or stroke you just right? Let me know below in the comments section or Email me at buelahman {AT} g m a i l {DOT} com

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All posts are opinions meant to foster comment, reporting, teaching & study under the “fair use doctrine” in Sec. 107 of U.S. Code Title 17. No statement of fact is made or should be implied. Ads appearing on this blog are solely the product of the advertiser and do not necessarily reflect the opinions of BuehlahMan’s Revolt or WordPress.com

Everyone Is A Pothead (They Just Don’t Know It)

Exercise, Manual Therapy, and the Endocannabinoid System: Why we’re all inherently potheads

by Dr. Andreo A. Spina

ths-ecs-for-ths2-1024x753

What if I were to tell you that this biological system permeates the entire human body with receptors located in skeletal muscle, the digestive tract, adipose (fat) tissue, and throughout the peripheral and central nervous systems (including the brain)? Again, you would question why this system is not studied, discussed, or even mentioned in most in physiology/health classes.

What if I were to tell you that the Endocannabinoid system (or ECS):

–   Helps regulate the central control of energy balance

–   Helps regulate metabolic processes (including storage)

–   Plays a key role in the maintenance of bone mass

–   Regulates intestinal motility

–   Promotes/regulates sleep

–   Is involved in neuromodulation and immunomodulation in the immune system

–   Is involved in modulating insulin sensitivity

–   Is involved in the regulation of pain signaling

–   And much more

You can read the entire, educational blog post here.

weed-slims

I have been preaching for quite some time that somehow or another (meaning that our bodies were either designed or we evolved to receive the cannabinoids found only in the cannabis plant),  this “endocannabinoid system” is found in our bodies (yes, each and every human being, including the naysayers, the prohibitionists and the ignorant).

What (you ask)?

“God wouldn’t create us to receive something that is evil, would He?”

I guess I am just supposed to believe that it is all a coincidence or not important or useful (or maybe God didn’t know what He was doing), right?God_made_weed,_man_made_beer

Doesn’t that strike you as strange? That our body is made to use this substance, yet you have a government and complicit media/health/law enforcement that continually use erroneous data and conjecture to keep you from using it?

Can you not see that the misdirection has purpose to stop good health and healing? Do you understand they spend $TRILLIONS of your tax dollars on deception and perversions used to arrest people so certain entities can make huge amounts of money?

They don’t want you to know about alternative means of administration (like eating raw cannabis that will, in no way, get you “high”)? But because the THC can not be administered without heating the stuff except via a trans-dermal patch (and, yes, the THC is beneficial in many ways), one can eat “consumables” (foods with it in it),  oils produced from it, smoke it, or better yet, vaporize it . Most of these means heat the THC and one can use its palliative agents for their ailments. The point is that one doesn’t have to risk any lung irritation to use THC.

Heavy smoking can, indeed, cause a range of lung issues, but moderate smoking does not (no matter what the “official line” boot-lickers tell you). These prohibitionists continually minimize its advantages and call it all sorts of names meant to brainwash you against it. What is sad is that many people (like a former commenter who believes that they are informed on a variety of subjects I discuss here), fall for the lies of the very ones they generally G-GodCreatedMarijvehemently disagree with, regurgitate those very lies, and discount its usefulness which is becoming more and more evident (except to those, like her, who are willingly ignorant).

It is bizarre how ignorant and strong-headed some self-described thinkers can be, when, in fact, they are simply regurgitating the tired old indoctrination.

On a side note, this desperate person continued to harass me via email after our last discussion on the blog (which culminated in me banning their IP address from commenting).

Her parting shot was,

“You’re certainly not an intellectual.  More like a pot-head with a fragile ego.”

She’s only half correct: I am no intellectual. But let me assure you that I have absolutely no cannabis in my system (altho I believe that it god_grows_marijuana1would help with my slightly elevated BP and arthritic aches and pains). As a matter of fact, there are NO drugs of any type in my system, except for my BP meds (hellfire, I don’t even drink alcohol, which is deadly, but legal). But from the last interaction with that bedlamite, it would seem that she is very well-versed in alcohol consumption.

My continual push for medical marijuana legalization is for people near and dear to me. I know people who suffer daily and would benefit from this medicine, but it is still illegal where I live (and many of the citizens still believe the dogma). Nevertheless, it is ludicrous to keep this innocuous wonder plant illegal for any reason, despite the nincompoops that still accept and trust the very people who profit from its illegality or want you sick and dying or are too brainwashed to know the difference.

What if God smoked cannabis?

What if God smoked cannabis?

It is high time (no pun intended) to understand that they LIE to us about this (and virtually everything else).

Now, enjoy some a collection of history’s best weed songs (I assembled in a playlist only available to readers here):

And if you haven’t done so, yet, please subscribe to the YT channel. Since they deleted my last channel, I hope to get more play on my videos again. Also note that I very much appreciate all the subscribers to this pitiful little blog. It is growing substantially (I know, only 500 or so, but that is big time for me)!

 

h/t Cannabis Journal (originally featured at Functional Anatomy Blog)

Follow @BuelahMan

BuelaHuh?

Did I rub you the wrong way or stroke you just right? Let me know below in the comments section or Email me at buelahman {AT} g m a i l {DOT} com

If for some reason you actually liked this post, click the “Like” button below. If you feel like someone else needs to see this (or you just want to ruin someone’s day), click the Share Button at the bottom of the post and heap this upon some undeserving soul. And as sad as this thought may be, it may be remotely possible that us rednecks here at The Revolt please you enough (or more than likely, you are just a glutton for punishment??), that you feel an overwhelming desire to subscribe via the Email subscription and/or RSS Feed buttons found on the upper right hand corner of this page (may the Lord have mercy on your soul).

All posts are opinions meant to foster comment, reporting, teaching & study under the “fair use doctrine” in Sec. 107 of U.S. Code Title 17. No statement of fact is made or should be implied. Ads appearing on this blog are solely the product of the advertiser and do not necessarily reflect the opinions of BuehlahMan’s Revolt or WordPress.com